Depression Prompts Blood Vessel Plaques
By editor | July 2, 2007
Depression a common term for a sad or low mood or the loss of pleasure; an emotion that does not affect capacity to perform personal and vocational obligations.
If depression has turned out to be your life companion then try to get rid of it as soon as possible because it may trigger the development of blood vessel plaques, known as atherosclerosis. Atherosclerosis is a condition, which experts believe may put you at increased risk of heart attack, stroke and several other cardiovascular health problems.
If depression has turned out to be your life companion then try to get rid of it as soon as possible because it may trigger the development of blood vessel plaques, known as atherosclerosis. Atherosclerosis is a condition, which experts believe may put you at increased risk of heart attack, stroke and several other cardiovascular health problems.
Researchers brought out this idea after a long study of three years, during which they evaluated the contribution of depression, anxiety, and anger to atherosclerosis among 324 men and women between 50 and 70 years old.
Briefing about these findings investigators concluded:
Our results indicate that depression, but perhaps not anxiety and hostility/anger, may be involved in the initiation and/or progression of atherosclerosis.
This is really a serious issue that has been undraped through this study, which clearly shows that how dangerous depression could be. Still, we can’t deny the fact that this is not the first study to show that depression is linked to bad health since previous studies have well promulgated this fact. Here is a look:-
- How Depression Affects the Workplace
- Cognitive functions in depressive disorders
- Depression Can Make Your Bones Brittle
- Anger, Depression, Hostility and Anxiety Leads to Heart Diseases!
- Stressed Moms May Slower Baby’s Growth In the Womb!
Atherosclerosis:
Atherosclerosis is a disease affecting the arterial blood vessel. It is a chronic inflammatory response in the walls of arteries, in large part to the deposition of lipoproteins (plasma proteins that carry cholesterol and triglycerides). It is commonly referred to as a “hardening” or “furring” of the arteries. It is caused by the formation of multiple plaques within the arteries.
Pathologically, the atheromatous plaque is divided into three distinct components:
These complications are chronic, slowly progressing and cumulative. Most commonly, soft plaque suddenly ruptures (see vulnerable plaque), causing the formation of a thrombus that will rapidly slow or stop blood flow, e.g. 5 minutes, leading to death of the tissues fed by the artery. This catastrophic event is called an infarction. One of the most common recognized scenarios is called coronary thrombosis of a coronary artery causing myocardial infarction (a heart attack). Another common scenario in very advanced disease is claudication from insufficient blood supply to the legs, typically due to a combination of both stenosis and aneurysmal segments narrowed with clots. Since atherosclerosis is a body wide process, similar events also occur in the arteries to the brain, intestines, kidneys, legs, etc.
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